Turkish Journal of Nephrology
Case Report

Can amantadine cause resistant hypernatremia in the treatment of hypoxic brain injury due to cardiac arrest?

1.

Gazi Yaşargil Training and Research Hospital, Division of Critical Care, Diyarbakır, Turkey

2.

Gazi Yaşargil Training and Research Hospital, Department of Neurology, Diyarbakır, Turkey

Turkish J Nephrol 2022; 31: 385-388
DOI: 10.5152/turkjnephrol.2022.211295
Read: 123 Downloads: 63 Published: 18 May 2022

Out-of-hospital cardiac arrest complicated by neurological deterioration provides worse prognosis. Therapeutic hypothermia is the primary treatment to indicate utility in improving survival as well as limiting neurological damage in cardiac arrest patients. A 56 year-old man patient, not to be treated with therapeutic hypothermia, received more than 45 minutes of cardiopulmonary resuscitation but significant damage as a result of hypoxic brain injury. We present a case of treatment-resistant hypernatremia, which was thought to be due to a neuroprotective agent used in hypoxic brain injury treatment and was not previously mentioned in the literature.

Cite this article as: Sami Kalın B, İhsan Sert A, Altun K, Öztürk Ü. Can amantadine cause resistant hypernatremia in the treatment of hypoxic brain injury due to cardiac arrest? Turk J Nephrol. 2022;31(4):385-388.

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